HANDS ON FESS & TEMPORAL BONE DISSECTION COURSE
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workshop secretariat
Dr.Rau's ENT super specialty hospital,
1/2rt, housing board colony, punjagutta,
Hyderabad - 500082
contact no’s - +919849085060, +919989225035
email - drgvsrao@raosentcare.com, drchaitanya@raosentcare.com
course coordinators - Dr.Krishna Reddy, Dr.Arun kumar, Dr.Anoop,
Dr.Chaitanya Rau.
Tuesday, 31 May 2011
Comment on Otoscopic Findings
Correct Responses
Dr Srinivas (Bangalore)
Dr. Prahlada N.B(Bangalore)
Dr. Rishi Gautam Aggarwal (Ambala)
Dr.Pankaj (Lucknow)
Dr. Deepak Dalmia(Mumbai)
Dr.Faizy (Cairo)
Dr.Rajeev Kapila(Ludhiana)
Dr.Venketasan (Chennai)
In some patients, an outwardly bulging, thin atrophic area or "herniation" of the tympanic membrane will be encountered. These “pulsion hernias” are asymptomatic and do not seem to interfere in any way with normal epithelial migration along the surface of the tympanic membrane.
There appear to be two prerequisites necessary for the development of a pulsion hernia.
The first is a preexisting defect in the fibrous middle layer of the pars tensa of the tympanic membrane.
The second is the presence of positive pressure within the middle ear, which forces the thinned portion of the tympanic membrane laterally into the canal (the pulsion).
If the fibrous middle layer of the pars tensa were intact, the tympanic membrane would be unable to herniated laterally to any significant extent.
The thinness of the pars tensa of the tympanic membrane over the pulsion pocket suggests that the herniation of the tympanic membrane occurred in an area where the fibrous middle layer of membrane has disappeared (e.g., the site of a previous, healed perforation or retraction pocket), leaving an area where the membrane consists of only two layers (a dimeric membrane).
Dr Srinivas (Bangalore)
Dr. Prahlada N.B(Bangalore)
Dr. Rishi Gautam Aggarwal (Ambala)
Dr.Pankaj (Lucknow)
Dr. Deepak Dalmia(Mumbai)
Dr.Faizy (Cairo)
Dr.Rajeev Kapila(Ludhiana)
Dr.Venketasan (Chennai)
In some patients, an outwardly bulging, thin atrophic area or "herniation" of the tympanic membrane will be encountered. These “pulsion hernias” are asymptomatic and do not seem to interfere in any way with normal epithelial migration along the surface of the tympanic membrane.There appear to be two prerequisites necessary for the development of a pulsion hernia.
The first is a preexisting defect in the fibrous middle layer of the pars tensa of the tympanic membrane.
The second is the presence of positive pressure within the middle ear, which forces the thinned portion of the tympanic membrane laterally into the canal (the pulsion).
If the fibrous middle layer of the pars tensa were intact, the tympanic membrane would be unable to herniated laterally to any significant extent.
The thinness of the pars tensa of the tympanic membrane over the pulsion pocket suggests that the herniation of the tympanic membrane occurred in an area where the fibrous middle layer of membrane has disappeared (e.g., the site of a previous, healed perforation or retraction pocket), leaving an area where the membrane consists of only two layers (a dimeric membrane).
Monday, 30 May 2011
E- BOSS 2011 ( ENDOSCOPIC BASE OF SKULL SURGERY )
THEME : LOOKING BEYOND THE SINUSES
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DATES: AUGUST 19-21,2011
VENUE:ROYAL PEARL HOSPITAL
C - 12 , 3D CROSS THILLAINAGAR
TRICHY
TAMILNADU
FACULTY
PROF AMIN KASSAM FROM OTAWA
PROF RICCARDO CARRAU FROM CA
PROF DANIEL PREVADILO FROM OHIO
CONTACT FOR WORKSHOP AND CADAVARIC DISSECTION
DR JANAKIRAM
PH 09842461176
CLICK HERE TO VIEW BROCHURE
Sunday, 29 May 2011
World No Tobacco Day,May 31
- approximately 120 million citizens of India are smokers (among the largest in the world)?
- the Indian tobacco industry sells 102 billion cigarettes every year?
- an estimated 240 million people in India use tobacco?
- 40% of deaths in India directly or indirectly occur due to tobacco intake?
- smoking is more prevalent among rural women, with 2% of them being active smokers, as compared to only 0.5% women smokers in urban areas in India?
- more than 11% Indian women consume tobacco products.
Schneiderian papillomas
The ectodermally derived ciliated respiratory mucosa that lines the sinonasal tract, so-called Schneiderian membrane, gives rise to three morphologically distinct papillomas. These are referred to individually as the fungiform(BOTTOM L), inverted(TOP), and oncocytic papillomas(BOTTOM R) or, collectively, as Schneiderian papillomas.
These benign lesions were named in honor of C. Victor Schneider who, in the 1600s, demonstrated that nasal mucosa produces catarrh and not CSF and identified its origin from the ectoderm.
Unlike the rest of the upper respiratory tract mucosa, the sinonasal mucosa is ectodermal in origin, derived originally from the stomodeum (ie, primitive mouth) in the fourth week of gestation. Sinonasal mucosa is continuous with the mucosal lining of the nasopharynx, which is of endodermal origin but is of identical histology.
Primary tuberculosis of submandibular salivary gland.
J Glob Infect Dis. 2011 Jan;3(1):82-5.
Primary tuberculosis of submandibular salivary gland.
Tauro LF, George C, Kamath A, Swethadri G, Gatty R.
Source
Department of General Surgery, Fr. Muller Medical College Hospital, Kankanady, Mangalore (D.K.), Karnataka, India.
Abstract
Tuberculosis of the submandibular salivary gland is a rare condition and only a few cases have been reported in literature. Tubercular sialadenitis is most frequently seen in immunosuppressed patients. Diagnosis of this disease is difficult. Although fine needle aspiration cytology is useful in diagnosis, excisional biopsy is often required. Polymerase chain reaction for mycobacterium tuberculosis is a reliable diagnostic tool, and if available, it should be performed before surgical intervention to enable differential diagnosis of a salivary gland tumor. We report two cases of the submandibular salivary gland tuberculosis from South India (Mangalore located in the coastal belt of Karnataka) that proved diagnostically challenging. Both patients responded well to antitubercular therapy and surgery was avoided.
PMID: 21572614 [PubMed - in process] PMCID: PMC3068584 Free PMC Article
READ MORE....
Primary tuberculosis of submandibular salivary gland.
Tauro LF, George C, Kamath A, Swethadri G, Gatty R.
Source
Department of General Surgery, Fr. Muller Medical College Hospital, Kankanady, Mangalore (D.K.), Karnataka, India.
Abstract
Tuberculosis of the submandibular salivary gland is a rare condition and only a few cases have been reported in literature. Tubercular sialadenitis is most frequently seen in immunosuppressed patients. Diagnosis of this disease is difficult. Although fine needle aspiration cytology is useful in diagnosis, excisional biopsy is often required. Polymerase chain reaction for mycobacterium tuberculosis is a reliable diagnostic tool, and if available, it should be performed before surgical intervention to enable differential diagnosis of a salivary gland tumor. We report two cases of the submandibular salivary gland tuberculosis from South India (Mangalore located in the coastal belt of Karnataka) that proved diagnostically challenging. Both patients responded well to antitubercular therapy and surgery was avoided.
PMID: 21572614 [PubMed - in process] PMCID: PMC3068584 Free PMC Article
READ MORE....
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